In the sliding filament theory of muscle contraction, the thin filament (actin) slides over the thick filament (myosin). Myosin is responsible for pulling the actin filaments towards the center of the sarcomere during muscle contraction.
The sliding filament model of contraction involves actin filaments overlapping myosin filaments.
M-line, causing overlap with the thick filament during muscle contraction. This results in the sarcomere shortening and overall muscle contraction.
During muscle contraction, the thin filaments (actin) are pulled towards the center of the sarcomere, which causes the Z-lines to move closer together. This process is facilitated by the interaction between actin and myosin filaments during the sliding filament mechanism of muscle contraction.
The thin filament of a myocardial cell is composed primarily of actin, tropomyosin, and troponin proteins. These proteins play a crucial role in regulating the contraction and relaxation of the heart muscle by interacting with the thick filament during the process of muscle contraction.
decreased width of the H band during contraction
Myosin is a protein that is not found in the thin filament. Myosin is a motor protein that is primarily found in the thick filament of muscle cells and is responsible for muscle contraction. The thin filament contains proteins such as actin, tropomyosin, and troponin.
filament
Actin
thick filaments
The sliding filament theory of muscle contraction was proposed by Andrew Huxley and Rolf Niedergerke in 1954.
The myosin myofilament pulls on the actin myofilament during muscle contraction. This interaction, known as the sliding filament theory, results in the shortening of the sarcomere and muscle contraction.